Maximilian Quertermous

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Publications

  • Loss of CDKN2B Promotes p53-Dependent Smooth Muscle Cell Apoptosis and Aneurysm Formation

    Arteriosclerosis, Thrombosis, and Vascular Biology (Journal of the American Heart Association)

    Genomewide association studies have implicated allelic variation at 9p21.3 in multiple forms of vascular disease,including atherosclerotic coronary heart disease and abdominal aortic aneurysm. As for other genes at 9p21.3, human expression quantitative trait locus studies have associated expression of the tumor suppressor gene CDKN2B with the risk haplotype, but its potential role in vascular pathobiology remains unclear. These results suggest that reduced CDKN2B expression and increased…

    Genomewide association studies have implicated allelic variation at 9p21.3 in multiple forms of vascular disease,including atherosclerotic coronary heart disease and abdominal aortic aneurysm. As for other genes at 9p21.3, human expression quantitative trait locus studies have associated expression of the tumor suppressor gene CDKN2B with the risk haplotype, but its potential role in vascular pathobiology remains unclear. These results suggest that reduced CDKN2B expression and increased smooth muscle cell apoptosis may be one mechanism underlying the 9p21.3 association with aneurysmal disease.

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Honors & Awards

  • 1st Place, J.P. Morgan National Investment Banking Case Challenge

    J.P. Morgan

  • James B. Angell Scholar

    University of Michigan

  • University Honors

    University of Michigan

  • AP Scholar with Distinction

    -

  • National Merit Commended Student

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